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Additionally, we evaluated the expression levels of sodium-taurocholate cotransporting polypeptide, which was found to be suppressed during chronic HBV infection.
We evaluated the expression levels of epithelial cell adhesion molecule and the in vitro activity of solitomab against primary uterine serous carcinoma cell lines in vitro and ex-vivo in the ascites of patients with uterine serous carcinoma.
We also evaluated the expression levels of the Cx43 protein, a marker of the working myocardium.
To this aim, we first evaluated the expression levels of TrkB and its primary ligand BDNF in the three cell lines.
Because previous reports have revealed a mechanism by which the action of insulin is inhibited as the result of the increased ubiquitination and degradation of IRS1 via SOCS1 and SOCS3 [20], we first evaluated the expression levels of SOCS1 and SOCS3 proteins in the transgenic mice and HBx-transfected cells.
We therefore evaluated the expression levels of locally produced IGF-I in the tumor.
Several studies have evaluated the expression levels of miR in MPM [ 10– 10].
We evaluated the expression levels of STX11 and UTRN using published data (GSE6338 and GSE19069).
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Therefore, we evaluated the expression level of key transcription factors involved in CD8+ T cell response.
To confirm the specific effect of CHX and WM treatment, we also evaluated the expression level of the most prominent isoform, SHOXa, which does not contain a PTC (Figure 3 A, middle graph, and 3 B, right graph).
We first evaluated the expression level of ERα and ERβ, β4 integrin subunit, ErbB2, and ErbB-3 in a series of human mammary tumor cell lines including MDA-MB 231, MDA-MB 361, SKBr3, BT474, BT549, and T47D.
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