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To test whether fetal activation of microglia could be sufficient to induce delayed synaptic defects, we induced prenatal pharmacological inflammation and evaluated the consequences on synaptic function.
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While numerous studies have documented short-term effects of prescribed burning on nutrient dynamics, few have evaluated the consequences of repeated fire on N availability and ecosystem productivity over longer time frames, such as a 3-year fire return interval.
There is no basis to judge which of these periods would be acceptable, since a few [ 17, 27, 29] of the studies evaluated the consequences of the delay on the outcome of TB treatment.
This study evaluated the consequences of late preterm birth on child growth in the first two years of life as compared to term deliveries in a population-based cohort from a middle-income country.
We next evaluated the consequences of AmphoB's altering pH on acid-induced HA activation.
We evaluated the consequences of disrupted kif5c-mediated mitochondrial trafficking on mitochondrial form and function in primary rat cortical neurons.
However, no study has evaluated the consequences of intermittent contraction with low blood flow on skeletal muscle tissue.
We next evaluated the consequence of such prenatal inflammation on glutamatergic transmission.
Next, we evaluated the consequence of the neuronal expression of Trap1 on phenotypes associated with muscle degeneration.
Therefore, we evaluated the consequence of fixation with 2% paraformaldehyde (PFA) on the FRET efficiency in our FACS assay.
The objectives of this study were to evaluate the consequences of aging on the temporal organization of the antioxidant defense system and oxidative status as well as to analyze the endogenous clock activity, in the hippocampus of aged rats.
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