To identify TFs contributing to aberrant KC activity, including abnormal differentiation and excessive proliferation, we evaluated gene expression in psoriasis lesions from a meta-cohort of 237 patients.
In silico analysis was performed through a PubMed search of articles with primer sequences that evaluated gene expression of C. albicans.
To understand the complex regulatory pathways that underlie the development of refractive errors, expression profiling has evaluated gene expression in ocular tissues of well-characterized experimental models that alter postnatal eye growth and induce refractive errors.
Following euthanasia, the tendons were harvested and DNA, hydroxyproline, and glycosaminoglycan content determined, mechanical strength and stiffness evaluated, gene expression and spatial arrangement of collagen types I and III assessed by northern blot and in situ hybridization, and tendon fiber architecture assessed by polarized light microscopy.
This study evaluated gene expression responses from the perspective of a PPI network and several external functional information sources.
Relatively few studies have evaluated gene expression changes of EHEC O157∶H7 in the context of an infection model.
Other studies employing microarray technology have evaluated gene expression profiling of E. coli in the context of phylogenetic studies [11] [14], [31] and in response to either different environmental growth conditions or varying growth media [15] [20].
In this study we have evaluated gene expression changes in erythroblasts that result from knockout of the antioxidant protein SOD2 in hematopoietic tissues a model previously shown to have many features of congenital or acquired sideroblastic anemia.
We have evaluated gene expression and histological changes associated with human tendinopathy.
We evaluated gene expression using the cycle threshold (Ct) value from each sample.
We also evaluated gene expression patterns and activated pathways associated with RANK and RANKL expression.
