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In the current study, using specific polyclonal antibodies against GalNAc-T3 and -T6, we evaluated expressions of GalNAc-T3/6 in RCC specimens with patients' clinicopathological data, showing that only GalNAc-T3 was expressed in most human RCCs, especially those with worse ECOG PS, or higher Fuhrman's grade and vascular invasion (VI).
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We then evaluated expression of bphD.
We also evaluated expression of CD25, an additional marker of activated and regulatory T cells [32].
We next evaluated expression and secretion of the T3sVHHs by Western blot (WB).
We also evaluated expression across the cortex gene set as a continuous variable.
We previously catalogued and evaluated expression of multiple splicing forms of TCF7L2 in several types of human tissue [21].
We have evaluated expression levels of candidate genes previously identified through genetic and protein interaction analyses [7].
Here, we evaluated expression and function of ASB9 in detail.
Next, we evaluated expression of Cnr1 and miR-26b to confirm their relative expression levels.
Here, we have evaluated expression levels of 8 titin ligands in endomyocardial biopsies (EMB).
A second limitation of this study is that we evaluated expression of extracellular EpCAM only.
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