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We used RNAseq to evaluate the transcriptome response in a Cx.
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To evaluate the transcriptome-wide effects of ETV2 expression in HSkMCs, we performed microarray studies on HSkMCs and ETV2-HSkMCs.
Accordingly, we next evaluated the extent to which the profile of miRNAs observed during hypoxia was consonant with mRNA expression (i.e., the transcriptome response).
Comprehensive analysis of the transcriptome response by microarray identified genes with a differential response to pulsatile versus continuous glucocorticoid delivery.
In contrast, the transcriptome response to hypoxia are completed after 5 6 generations; and the transcriptome response to reoxygenation are completed in 2 generations [ 21].
The superordinate principles governing the transcriptome response of differentiating cells exposed to drugs are still unclear.
We analyzed the transcriptome response of yeast to toxic concentrations of selenite.
Consequently, the transcriptome response to competition might be difficult to interpret.
However, there is no information on the transcriptome response triggered by UV-B in grapevine.
The independence of the transcriptome response from the presence of drugs suggested that the transcriptome changes did not inform on direct drug signaling.
Thus, it is unlikely that the transcriptome response at late time points was triggered by a direct response to HDACi.
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