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The term 'resistant' can be applied to those individuals within a single snail species that are able to evade infection by a species or strain of schistosome that is normally capable of parasitizing that species of snail.
Several of the G. pallida effector gene families are considerably expanded and this may be a reflection of selection pressure to avoid detection or maintain function within a host that is under selection pressure to evade infection.
Folding over the protruding end of the diaper, or cutting out a hole beforehand can help evade infection.
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Nevertheless, plants can detect and evade most infection attempts through constitutive and inducible immune responses.
It is likely that the mutator bacteria evolved very quickly to evade phage infection and the phages were unable to persist.
Indeed, the complement system acts as a physiological barrier during the infection which parasites have to evade to successfully infect the host.
HBV infection regulates the apoptotic machinery to establish persistent infection and evade the immune system.
Using Evade in Infection is a great way to get more humans.
CD8+ CTL (cytotoxic T-lymphocyte -derived IFN-g may be especially imporT-lymphocyte -derivedacking MHC class II molecules, e.g. in T-lymphocyte -derivedophages where mycobacteria can evade recognition during chronic infection by sequestering their antigens away fromayensitized CD4+ T cells.
The complement system, therefore, acts as a physiological barrier which resistant strains have to evade for successful host infection.
Effectors selected to facilitate infection and evade recognition by R genes in one host might be recognised by the R genes in another host and be maladaptive.
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