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Increased "false memory" errors and accompanying bilateral prefrontal activation in schizophrenia suggest that the etiology of memory errors must be considered when comparing group performances.
The lack of consensus regarding the etiology of memory concern, 61 and its association with psychopathology, has reinforced the notion that concern is purely subjective.
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However, our results suggest that such analyses provide novel insight into the etiology of working memory deficits in schizophrenia.
It has been suggested that if memory of pain itself is a primary etiology of chronic pain, memory loss could result in pain reduction or pain-free state [15].
Ashraf and Kunes hypothesized that miRNAs might serve as the molecular etiology for memory and presented evidence for RISC-mediated control of RNA stability underlying protein synaptic protein synthesis associated with memory [32], [46].
So it has been suggested that if pain memory itself is a primary etiology of persistent pain, how to inhibit the memory of pain become an effective way of analgesia for prolonged pain.
No one model or conceptualization has gained primacy in this area, but cognitive models of the etiology of PTSD have attempted to explain the memory abnormalities seen.
Memory, all kinds of memory, is exploding.
The obliteration of memory.
Feat of memory?
The floodgates of memory open.
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Since I tried Ludwig back in 2017, I have been constantly using it in both editing and translation. Ever since, I suggest it to my translators at ProSciEditing.

Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com