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The etiology for the development of IUAs also appears to impact outcome.
Our study suggests a complex etiology for the development of psychological disorders after ICU discharge, as both patient characteristics, severity of disease, and ICU-related variables represented risk factors for psychological dysfunction after critical illness.
In recent studies, an infectious etiology for the development of Charcot neuropathy (CN) has been proposed [ 3].
Thus, hypotension in combination with coagulation derangement might be the possible etiology for the development of AKI.
The resultant pulmonary endothelial cell injury leads to a decrease in the production of PGI2, which might be an important etiology for the development of acute respiratory distress syndrome, a critical complication of sepsis.
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The understanding of its underlying etiology is essential for the development of novel effective therapies.
Therefore, an understanding of the underlying etiology is essential for the development of novel effective therapies for NPC.
An extensive research program is needed to understand the extent of the problem, its underlying etiology, and the development of a strategy for prevention and intervention.
The etiology for PCa development and molecular mechanisms underlying castration resistant progression are incompletely understood.
Our study provides important insights for the future investigation of molecular functionality related to PrCa etiology and the development of PrCa diagnosis tools and targeted therapy strategies.
Different etiologies have been described for the development of large defects after cleft palate repair like tension of the wound closure related to the surgeon's performance and cleft width, infection, and hematoma formation; however, it appears that necrosis of the mucoperiosteal flap is the most common cause of this complication [ 29].
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