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Mean PMC activation magnitude parameter estimates, at baseline, were negative in the control (−0.57±0.12) and MCI-Nonconverter (−0.33±0.14) groups, and positive in the MCI-Converter (0.37±0.40) and AD (0.92±0.30) groups.
Geometric mean estimates at baseline were calculated from raw data.
This analysis resulted in separate slope estimates at baseline for (i) BMI gainer, obese patients; (ii) BMI gainer, non-obese patients; (iii) non-BMI gainer, obese patients; and (iv) non-BMI gainer, non-obese patients.
Similarly, all objectively measured time estimates at baseline were significantly correlated with their corresponding time estimates at follow-up (r = 0.61 to 0.63, P < 0.0001), indicating a high degree of stability of patterns of physical activity.
In this model the mean utility difference between the progression-free state (P0) and post-progression state (P1), at −0.041util, is smaller than that observed between the point estimates at baseline (P0.1) and first post-progression observation (P1.1), but of threshold statistical significance (p = 0.051).
Not surprisingly, these individuals had GFR estimates at baseline that were close to the 60 mL/min per 1.73m2 threshold used by clinical guidelines to define stage 3 CKD and were disproportionately women when compared to those with eGFR sustained < 60 mL/min per 1.73m.
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SOFA score was estimated at baseline and daily until discharge, death or up to 28 days.
Hematological parameters were estimated at baseline and end-study.
Reliabilities were estimated at baseline and at Week 12.
Usual dietary intake was estimated at baseline from study-specific FFQs.
Individuals' nutrient intake and nutritional risk score were estimated at baseline (1984-1988) and follow-up (1992-1996).
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