Several essential cellular functions of mitochondria depend on a high degree of functional interaction between the nuclear and mitochondrial genomes.
This is consistent with the essential cellular functions of DNA replication machinery, and is further suggestive of functional domains underlying these specific single mutations.
Adenosine nucleotides provide energy for many essential cellular functions.
Their topological importance is consistent with an important role in terms of essential cellular functions.
Beside their essential cellular functions, isoprenoids have value as pharmaceuticals, nutriceuticals, pesticides, and fuel alternatives.
Protein kinases are an important class of proteins which are involved in various essential cellular functions, including signaling, growth, development and homeostasis [1 3].
The interplay of the proteostasis network with other essential cellular functions such as cilia and vesicle transport is only beginning to be unraveled [3].
Mitochondria participate in a large number of essential cellular functions.
MicroRNAs play a critical role in many essential cellular functions in the mammalian species.
Therefore, it is conceivable that evolution would select for an overall decreased aggregation propensity in operons performing essential cellular functions.
This finding suggests that the regulation of dFoxO translation by IRES may help retain essential cellular functions during Drosophila fasting.
