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These interventions have been identified as essential cardiovascular disease interventions that need to be financed as a first step to universal coverage.
This implies that, in isolation, the role of hypercholesterolemia in the incidence of cardiovascular events is neither essential (cardiovascular events occur in the absence of hypercholesterolemia) nor sufficient (most patients with high cholesterol do not have cardiovascular events) [ 4].
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"It's essential that cardiovascular screening and prevention programmes don't get cut as a result".
These studies identify the calcineurin/Rcan1-signaling cascade as a potential therapeutic target through which to benefit from innate circadian changes in cardiac protection without disrupting core circadian oscillations that are essential to cardiovascular, metabolic, and mental health.
Choline is essential for cardiovascular and brain function.
Posttranslational protein arginylation mediated by Ate1 is essential for cardiovascular development, actin cytoskeleton functioning, and cell migration.
Future studies should aim at establishing age- and gender-specific normative values which are essential for cardiovascular risk stratification.
Ate1 plays a role in the regulation of cytoskeleton and is essential for cardiovascular development and angiogenesis capillary remodeling driven by in-tissue migration of endothelial cells.
Vasopressin is essential for cardiovascular homeostasis.
α-pv is essential for cardiovascular development in mice.
ADH is essential for cardiovascular homeostasis and release from the hypothalamus [ 7].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com