Properties of hard nanocomposite coatings, especially hardness, can be explained by their nanostructure.
The two different periods in the Mediterranean surface forcing and especially their abruptness are explained by Demirov and Pinardi [46] with phase matching of long-term variations in NAO (in particular its intensification at the end of the 1980s), the Pacific North American Pattern, and the Eurasian Pattern.
The variation in these findings, especially in specificity, might be explained by the differences in included studies, since these recent publications additionally included studies with smaller populations and lower MRI field strengths.
The raised tail of the SFS may be explained by ASM, especially if the SFS is already left-shifted.
This result (especially the former) can be explained by the compromised health status of the non-Swiss population (see, for instance [ 25]) that leads to reduced HRQOL.
The rather high level of involvement in slot machine gambling, especially in Finland, could be explained by easy access and abundant availability.
Our evidence for strain-specific miRNA expression could imply that eQTLs, which have been observed at the level of expression of protein-coding genes, could be partially explained by miRNAs, especially when linked regions span large genomic distances.
Although some diseases associated with dysregulation of epigenetic modification at some genomic region have been found, EWAS has similar drawbacks to GWAS: it is difficult to estimate how much variation in phenotypes, especially for complex traits, is explained by epigenetic polymorphisms, even if there is evidence that they contribute to phenotypes, either biologically or statistically.
Similar findings have been reported in Germany and in USA, showing that adiponectin values in post-pubertal children are lower than in pre-pubertal children, especially in boys, which is explained by the elevated serum androgen levels in the former [ 20, 21].
As the documented decrease in cell proliferation cannot be totally explained by apoptosis, especially after RPS19 depletion, we studied cell cycle progression of erythroid cells derived from primary CD34+ cells of two RPL11+/Mut DBA patients and following RPS19 and RPL11 depletion in normal CD34+ cells.
