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The new agreement, still sub ject to approval by stockhold ers of both companies, calls for an exchange of eight shares of Volt common stock for one share of Teleprompter common.
One sees that, at ENZ point, ERs of both modes near the maximum, and ΔER is less than 0.01 dB/μm.
NITO for the maximum ER is near the ENZ point and NITO for the maximum ERs are at the ENZ point, for example, NITO = 6.0× 1020 cm− 3, the maximum ERs of both modes are 1.65 and 1.56 dB/μm at the wavelength of 1.50 μm, and the minimum ΔER is 0.009 dB/μm at the wavelength of 1.55 μm, which is our operation wavelength.
After 48-hr culture in the standard medium, ERs of both cell types decreased, whereas PR expression was mainly reduced in SCs.
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The Eox and Ered of both photoredox pair components were determined from cyclic voltammetry measurements (Table 1).
Depending on the cell type and mode of induction of ER stress, both the ubiquitin/proteasome pathway and the lysosome/autophagy pathway are stimulated in response to ER stress [ 5].
Furthermore, FACS sorted CD44+CD24−/low cells expressed barely detectable levels of ER, both when isolated from control and MCF-7TamR cultures (Fig 4E).
Deletion of the signal peptide did not prevent the binding between UFM1 and UFBP1 in vitro but it prevented the ER localization of both UFBP1 and UFM1.
There did not appear to be any considerable effect of E2 on the ER binding of both cell types.
As expected, ER exit of both CD4-TLR3 and CD4-TLR7 reqUNC93B1NC93B1, which is consistent with defective TLR3 and TLR7 signaling in Unc93b13d/3d mice (Tabeta et al., 2006), whereas CD4-TLR4 traffindependentlydently of UNC93B1.
Concerning the immunohistochemical expression of ER and PR, both the intensity (negative, 1+ to 3+) and percentage of immunopositive cells were evaluated.
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