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The Human Immunodeficiency Virus type 1 (HIV-1) is a highly pathogenic, evasive and difficult to eradicate agent that causes Acquired Immunodeficiency Syndrome (AIDS).
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Despite several studies demonstrating the susceptibility of B. mallei to numerous antibiotics in vitro, many failed to eradicate the agent from the organism of laboratory animals when challenged [ 1, 3, 4].
Its role is to eradicate foreign agents.
With vaccines and effective treatments often unavailable, the immune system's efforts to eradicate infectious agents or infected cells are frequently the only means to combat them.
1 One may speculate that similar to tuberculosis, the immune system, after its initial response to a triggering microorganism, is successful in eradicating the agent and the immune response subsides.
This response is effective in eradicating infectious agents such as viruses [ 3].
However, immune system fails to eradicate this causative agent, which can lead to the gastric mucosa pathological changes [ 1].
Hence, we investigated the utility treating infected mice with rifampicin at doses that limit toxin expression followed by gentamicin at bactericidal doses to eradicate the bacterial agent.
If the cause of the pain in the testicles is infection, specific antibiotics are given in order to eradicate the causative agent.
When occurring, it poses the double challenge of eradicating the infectious agent while preserving the articulation and its function.
When occurring, it poses the double challenge of eradicating the infectious agent while preserving the articulation and its function [2, 3, 5].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com