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Thus, for a chemical reaction at equilibrium, the amount of reactants being converted to products per unit time is exactly matched by the amount being converted to reactants (from products) per unit time.
In equilibrium, the amount of deposits created by banks has to be equal to the amount desired by depositors.
Quantification of the removed Pb2+ from probe solutions was performed by a UV spectrophotometric method as proposed by Krzek et al. Results suggest that at equilibrium, the amount of sorbed Pb II) increased as the probe solution also increased (gradually from 10 to 200 ppm).
Binding of 3R to 4R tau monomer or polymer may likely occur and slow 4R tau assembly, but at equilibrium the amount of assembled 4R tau would not decrease unless the 3R binding was irreversible.
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After this equilibrium time, the amount of heavy metals adsorbed did not significantly change with time.
After the equilibrium period, the amount of adsorbate nearly unchanged with time.
Essentially, the drug concentration is in equilibrium, where the amount metabolized to inactive metabolites and/or eliminated is the same as the amount of drug administered.
The reason for this is, in part, that most of the CNV analysis has been based on CNVs that are "tagged" by GWAS SNPs and thus covering only a small well-behaved genomic regions (in Hardy-Weinberg equilibrium), whereas the amount of "structural dark matter" remains relatively untouched by arrays or sequencing of those genomic regions that are not amenable to GWAS arrays (48).
In the dark a very small amount of carriers are generated because of above band gap blackbody radiation, and in equilibrium the same amount of carriers must recombine (the principle of detailed balance).
The time required to attain this state of equilibrium is termed the equilibrium time, and the amount of dye adsorbed at the equilibrium time reflects the maximum adsorption capacity of the adsorbent under those operating conditions (Bello et al. 2010).
This fact implies that lack of HDAC1 promotes binding of APPL1 to NuRD via HDAC2, further pointing to the equilibrium between the amounts of complexes containing APPL1 HDAC1 and APPL1 HDAC2 NuRD present in cells under normal conditions.
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