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The fitness in the ancestral environment of populations evolved under continuous exposure to atrazine was significantly higher than in the populations evolved in continuous exposure to glyphosate (T10 = 3.95, P < 0.01) or carbetamide (T6 = 3.598, P < 0.05).
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Now it was at last placed to fulfil its promise: to address global problems of the environment, of population, of women, of development and human rights, and above all, to help build an institutional framework for international peace and security.
But even in the constant environment of population cages, such as those used to maintain the stocks from which the Agg and Neut lines were derived, a great deal of genetic variation in aggressive behaviour can be maintained after many generations in the lab[1].
Similar cross-sectional epidemiological studies were conducted in Cotonou [ 13], Port-au-Prince (Haiti) and among the Haitian community of Montreal [ 14] to highlight the vastly different risk profiles based on the highly contrasting environments of populations with a common genetic heritage.
The fitness in the ancestral environment of all populations was compared to source populations and to populations that underwent continuous exposure using a Dunnett's corrected paired t-test, except when some of the populations in a regime did not evolve resistance, in which case Dunnett's corrected t-test was used.
129– 131 The origin of these epidemics is linked to sociopolitical changes and conflicts that have resulted in sudden modifications of the environment, displacement of populations, changes in human practices, and feeble or absent HAT control programs in a weak health care system environment.
The four mechanisms that were felt to have the greatest impact on public health were changes to the physical environment; movement of populations, pathogens, and trade; agriculture; and urbanization.
The general framework starts with an invisible change in the environment of the populations concerned.
We conclude that duplicates are a key source of variation, enabling genome-wide adaptation to the environment of yeast populations.
Since A. thaliana flowering variation has been involved in climatic adaptation [ 30] we analyzed if the similarities observed between Morocco and Iberia for flowering traits and genes might be determined by the climatic environment of local populations.
Introductions of non-native species can significantly alter the selective environment for populations of native species, which can respond through phenotypic plasticity or genetic adaptation.
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