Sentence examples for enteric cells from inspiring English sources

There are two main types of exotoxins produced by Clostridium: enterotoxins, which exert their actions on the enteric cells of the gastrointestinal tract, and neurotoxins, which cause neuronal dysfunction.

In particular, the surface layer (S-layer) mediates adhesion to enteric cells (2 ), but other functions have been proposed for this S-layer structure: it may act as a molecular sieve, protect against parasitic attack, or be a mechanism to evade the host immune system (3 ).

The lack of an effect of NT-3 in this system was somewhat unexpected, since it has been shown to promote survival of both ageing myenteric neurons in isolated plexus samples [8] and young enteric ganglion cells in dissociated cell culture [9], when these preparations were exposed to menadione- and hydrogen-peroxide, respectively.

We also found that the cellular receptor for CBV, CAR, is expressed both in enteric epithelial cells and in nerve cells within myenteric ganglia.

These results indicate that GDNF, but not NT-3 can promote increased expression of the antioxidant enzymes catalase and Cu/Zn SOD by enteric ganglion cells in this dissociated cell culture model.

They propose that the presence of acute and chronic inflammatory states, loss of enteric CD4T cells, and increased T-cell apoptosis and proliferation that occur in pathogenic SIV infection are the determining factors in disease progression.

The requirement for LGI4 for embryonic enteric glial cell and satellite cell proliferation contrasts with that of LGI1 for glioma cells as LGI1 inhibits proliferation of glioma cells (Chernova et al., 1998; Krex et al., 2002).

Fatty acid binding protein 7 (FABP7) is a marker of immature enteric glial cells, whereas S100 is expressed only by mature glial cells.

Both astrocytes and related enteric glial cells are able to induce barrier properties in gut epithelial cells, and S-nitrosoglutathione (GSNO) has been identified as one molecular mediator of this effect [ 208].

In addition, both astrocytes and related enteric glial cells are able to induce barrier properties in gut epithelial cells and the molecule GSNO has been identified as a molecular mediator of this effect [ 208].

CNS: Central nervous system; EGC: Enteric glial cells; ENS: Enteric nervous system; FAK: Focal adhesion kinase; IEB: Intestinal epithelial barrier; IEC: Intestinal epithelial cells; PPARγ: Peroxisome proliferator-activated receptor gamma; TGF-β1: Transforming growth factor beta-1; VIP: Vasoactive intestinal peptide.