Several studies indicated that exercise induced an upregulation of the BDNF in the hippocampus and thus could play an important role in the enhancement of cognitive function in humans [ 45].
Endogenous ketoacids produced during fasting have been proposed to regulate VGLUT function through blocking chloride ion-mediated allosteric enhancement of transporter function 276.
This study is the first to demonstrate that it is possible to induce skin intraepidermal nerve fiber regeneration accompanied by enhancement of neurovascular function, translating into improved symptoms as well as sensory nerve function.
A key motive for this non-medical use of neuropharmaceuticals is the enhancement of cognitive function in healthy individuals beyond normal human capacity [ 3- 5].
In a dystrophic animal model, punctual depletion of Tregs during the repair process increased muscle damage; similarly, enhancement of Tregs function decreased the pathology, indicating a crucial role for local Tregs in muscle damage and repair.
Although ATPase activity is closely associated with the function of Pgp1, an increase in ATPase activity does not necessarily lead to an enhancement of Pgp1 function.
In addition, various functions of KRG have been reported, e.g. anti-inflammatory, −coagulating, and -oxidative actions, enhancement of sexual function, and vasodilation [ 4– 8].
Many reports have shown the enhancement of ER function in a variety of human cancers, but the mechanism for the cancer cell-specific regulation of ER function remains unresolved.
An enhancement of dopaminergic function could in principle directly modulate incentive salience or alternatively enhance the predicted hedonic utility associated with the vacation destinations, which in turn could boost incentive salience.
In addition, it has been used by many individuals for its purported enhancement of memory function, which has been studied in hopes of finding a treatment or preventative for Alzheimer disease.
