"enhancement of damage" is correct and can be used in written English.
You can use this phrase to describe a situation when damage or harm has increased or become worse. For example: "The enhancement of damage caused by the storm was made much worse due to flooding in the area."
Contrary to this, an enhancement of damage efficiency is observed in the lighter two binary targets, while a rather weak reduction is observed in gallium arsenide.
The aim of introducing ductile metal interlayer was to compensate the low toughness of composite layers and consequently enhancement of damage tolerance of bundled structures along with prevention of their catastrophic failure through activation of extrinsic toughening mechanism.
This finding is in agreement with the conclusions of Mandraju et al (2008) who show distinctly different roles for the two isoforms of topoisomerase II, namely that although topoisomerase II α upregulation is linked to enhancement of damage to DNA during treatment with H2O2 (possibly by relaxing and opening chromatin structure), it is only topoisomerase II β that is involved in DSB rejoining.
Phospho-H2AX is a marker of dsDNA damage, but as this effect was relatively minor, it suggested that enhancement of DNA damage was not the main cause of the synergistic increase in cell death.
In particular, the nicotinic AChR antagonist showed a marked enhancement of brain damage compared to the saline controls (p < 0.01).
However, long-chain n-3 polyunsaturated fatty acids induce an immunosuppressive status that leads to a reduction of the host natural resistance to infectious agents as well as to an enhancement of oxidative damage.
In the absence of irradiation, GNP treatment results in a 30% enhancement of DNA damage foci in MDA-MB-231 cells compared to a 34% enhancement at 1 hour post irradiation suggesting the observed enhancement post irradiation is due to an extension of the original damage caused by GNP treatment and not an additive effect of radiation exposure.
No significant enhancement of CY damage occurred in either case.
Radiation-induced cell death is inhibited following galectin-1-mediated H-Ras activation and the subsequent enhancement of DNA damage repair.
Chronic administration of MISO for an 8 h period gave no enhancement of melphalan damage, whether melphalan was given half-way through or at the end of the period of dosing.
Some mechanisms of cisplatin resistance include reduction of platinum accumulation by alteration of transmembrane pumps, enhancement of DNA damage repair, and reduced apoptosis induction (Krishan et al, 1997; Cvijic et al, 1998; Ohmichi et al, 2005; Liu et al, 2007).
