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Treatment of mice with resveratrol enhanced the expressions of DR4 and DR5.
Since co-treatment of SAMC and CCl4 enhanced the expressions of antioxidant enzymes, reduced the nitric oxide (NO -dependent oxidative stress, aNO -dependentlipid peroxidative induced by CCl4.
Foxc2 overexpression not only increased the numbers of mineralized nodes and ALP activity, but also enhanced the expressions of Runx2, OCN, VEGF and PDGF-β in transfected BMSCs after osteogenic induction.
Moreover, the ligands of TNF superfamily such as FasL, CD40L and TNF-α were significantly up-regulated and enhanced the expressions of apoptosis-related cysteine peptidases, including caspase-10, -8, -7 and -3.
We found that IL-6 increased lung cancer cell chemotherapeutic resistance and enhanced the expressions of Bcl-2, Mcl-1, Bcl-xl and ABCG2 by activating the ATM/NF-kappaB pathway.
Furthermore, TM dose-dependently enhanced the expressions of PFK-1 and PGC-1 α in the liver of exercised mice, and the similar results were noted in ABM-treated group (P < 0.05, Figure 4).
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Moreover, exogenous application of ABA also specifically enhanced the expression of OsAPX8.
These results clearly showed that TIP60 as a downstream molecule of UHRF2 enhanced the expression of H3K9ac and H3K14ac.
In addition, RCPS enhanced the expression of CD40+, CD80+ and CD86+ on the dendritic cells (DCs).
Application of ASA under chilling stress enhanced the expression of CBF3 together with the COR47 gene (Fig. 7b).
In addition, SFAR4 enhanced the expression of genes involved in fatty acid metabolism during seed germination and seedling development.
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