Sentence examples for enhanced lipid content from inspiring English sources

Morphometric evaluation of cytoplasmic ORO staining showed that macroH2A1.1-overexpressing Hepa 1-6 or HepG2 cells were protected from lipid accumulation as compared with control cells transfected with an empty vector, while macroH2A1.2-overexpressing cells only slightly enhanced lipid content.

Thus, optimization of the light intensity provides a direct means of obtaining higher cell densities and enhanced lipid content.

Previous studies have focused on the culture of microalgae and enhanced lipid content of microalgae cells [ 22– 25], and conversion of microalgae oil to biodiesel [ 26– 29].

To date, significant advances in microalgal genomics have been achieved [ 6, 27], and two reports of enhanced lipid content of diatoms due to genetic engineering have been published [ 16, 17].

GmMYB73 overexpression enhanced lipid contents in both seeds and leaves of transgenic Arabidopsis plants.

Considering that GmMYB73 enhanced lipid contents in both seeds and leaves of transgenic Arabidopsis and Lotus plants, we further examined whether GmMYB73 could elevate lipid accumulation in transgenic hairy roots of soybean plants.

However, enhancing lipid content remains a major scientific challenge.

Therefore, using high salinity stress to stimulate lipid accumulation of microalgae and maintaining a satisfactory cell growth rate (for example, by enhancing adaptation to salinity) seems to be a promising strategy to enhance lipid content and lipid productivity simultaneously.

Many studies indicated that ME plays a key role in lipid biosynthesis: in Mortierella alpine the ME activity was in correlation with lipid accumulation [ 45], and the overexpression of genes encoding ME in M. circinelloides could enhance lipid content 2.5-fold [ 46].

We also found that two soybean transcription factor genes GmDof4 and GmDof11 enhance lipid content in seeds of transgenic Arabidopsis plants, through upregulation of lipid biosynthesis-related genes and downregulation of storage protein gene by direct binding to their promoter regions [ 43].

The oxidative renal injury is both dose-dependent and time-dependent, as evidenced by renal anti-oxidant depletion, enhanced lipid peroxidation, platinum content, plasma creatinine BUN, and blood urea levels in rat models [ 8].