The possible value of using educational activities, materials and contents to enhance hypothesized benefits accruing to direct contact also requires investigation.
While in cki1Δdpl1Δeki1Δ accumulation of diacylglycerol (DAG) not used as a substrate for PE/PC formation through the Kennedy pathway was not detected and TAG formation was not enhanced, we hypothesized that the reduced level of PE as a donor of fatty acids might have a negative impact on Lro1p catalyzed TAG synthesis.
We hypothesized enhanced connectivity between the hippocampus and cortex during scene construction, reflecting the need for the hippocampus to access cortically stored scene elements to facilitate construction.
Since several mutants in domain I appeared to display enhanced activity, we hypothesized that the corresponding residues mediate interactions with heme, thus leading to the increased release of the cytochrome c product (see Figure 1A, step 4) when mutated.
For both, the trigeminal chemosensory as well as the electrical cutaneous channels we could hypothesize enhanced neuroticism in people with high sensitivity to potentially painful and/or dangerous stimuli, both conceptually and also on the basis of some, albeit limited, evidence.
Because sufficient concentration of intracellular tRNA and optimized codons of recombinant proteins enhanced translation efficiency, we hypothesized that sufficient supplement of host's tRNA improved protein productivity in mammalian cells.
Based on our finding that TLR3 expression is enhanced by RA, we hypothesized that RA could cooperate with TLR3 agonists to enhance their anti-cancer action.
Activation of the tumor stroma, which can include fibroblast activation, can contribute to enhanced metastasis and is hypothesized to mediate much of the downstream immune, angiogenic, and biophysical activation of the tumor microenvironment.
Since cardiac MuRF2−/− mice exhibited enhanced PPAR activity, we hypothesized that the induction of diabetic cardiomyopathy would result in both enhanced PPAR activity, resulting in significant cardiac dysfunction compared with wildtype mice.
This study was motivated by the hypothesis that application of thyroid hormones to articular chondrocytes in an in vitro model of scaffold-free cartilage regeneration would induce matrix maturation and enhance matrix properties; in particular, sequential application of T3 and PTH was hypothesized to enhance neocartilage properties without hypertrophic marker expression.
