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Already very early, the synergy between extra-framework aluminium, Lewis acid sites formed due to partial release of aluminium from the framework upon hydrothermal treatment and Brønsted acid sites was suggested.[ 1] Enhanced acidity of the catalyst was explained by direct coordination of Lewis acid sites to the Brønsted acid site.
The most obvious difference is the enhanced acidity of phenols.
Formation of more and more oxygen-containing groups upon the subsequent oxidation steps, detected by IR spectroscopy and elemental analysis, resulted in the enhanced acidity of graphite oxide.
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Furthermore, removing the EFAL species by acid leaching, which can lead to enhanced acidity through the formation of Lewis acid sites, reduced the catalyst activity.
However, the aluminium atoms which have been removed remain in the zeolite as EFAL species, which can lead to enhanced acidity through the formation of Lewis acid sites [23].
NH3-TPD and toluene-TPD results both exhibited an enhanced acidity and π bond adsorption ability of FeAlPO4-5 toward AlPO4-5 and the impregnated sample Fe2O3/AlPO4-5 Fe2O3/AlPO4-5
As a result, a comparatively small population of conformers with enhanced acidity can dominate the observed hydrogen exchange behavior.
In addition to enhanced acidity and double hydrogen bonding, the stacking interactions of the p-toluenesulfonyl ring in 10 with the electrophilic aldehyde are proposed to stabilize the transition state.
It is suggested that this extralattice species contributes to enhanced acidity.
The results show that the interactions between sulfates and NiO CeO2 ZrO2 solid solution lead to an enhanced Lewis acidity of the catalyst due to the electron withdrawing effect of sulfates mainly on Nin+.
This increase in biliary acidity also contributes to enhanced rates of apoptosis.
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