Sentence examples for engaging death from inspiring English sources

Cytotoxic lymphocytes (CTLs), the key players of cell mediated immunity, induce apoptosis by engaging death receptors or through exocytosis of cytolytic granules containing granzyme (proteases) and pore-forming protein (perforin).

Gaining Attention In the 1960s and '70s, Dr. Kevorkian shelved his quixotic campaign to engage death for social purposes and pursued a largely itinerant career as a medical pathologist.

Björnstrand played the role of the befuddled husband in Bergman's comic masterpiece Sommarnattens leende (1955; Smiles of a Summer Night) and the squire of the knight who engages Death in a game of chess in Det sjunde inseglet (1957; The Seventh Seal).

It has been proposed that when engaged, death receptors such as Fas and TNFR recruit and activate caspase-8, which can then cleave Bid into its active, truncated form tBid. Therefore, we examined tBid production in NPCs treated with LPS-activated MCM.

By engaging programmed death 1 (PD-1) on activated T cells, PD-L1+ tumors directly render tumor-specific T cells, including adoptively transferred T cells, functionally exhausted.

#Of the 47 men and 7 women not engaging in death talk at first interview, 17 men and 2 women booked additional interviews.

For instance, T-cells express a TNF-like weak inducer of apoptosis (TWEAK) which can stimulate HPC proliferation by engaging specific death receptor pathways.

E2F1 has a putative oncogenic signaling in DLBCL in the current series by orchestrating and engaging cell death pathways either alone or in cooperation with cellular proliferation pathways.

Our results show that direct signals from the TCR program T cells to die through Bim, which is fundamentally different from the secretion of death cytokines such as FasL that engage external death receptors.

Talking of Death Cafe London he says: "The time is right to offer a place where people can come to engage with death.

TRAIL is a member of the TNF superfamily that induces tumor-selective cell death by engaging the pro-apoptotic death receptors DR4 and DR5.