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Concerning the CEL volume as a secondary endpoint, we found a similar pattern: multivariate analysis revealed a significant reduction in CEL volume compared to baseline in the analyzable population as a whole (p = 0.008).
At study endpoint, we found 23 patients still in UWS based on behavioral assessment on at least two different occasions.
When using NHG as the endpoint we found a statistically significant association with T3 only in grade II.
By monitoring this endpoint, we found that TLR4 transcript levels increased during the first several hours of SMF exposure.
Although not a primary endpoint, we found an interesting and previously unreported association between hyponatremia and leukocyte count in these patients.
In analyzing our primary endpoint, we found that there was a secular trend associated with a natural decline of the time under MV (secular trend β1 = −0.055, P = 0.012).
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When we used sickness benefit and disability pension as endpoints, we found large differences between cancer survivors and women without breast cancer.
While it remains uncertain whether early IT is associated with a reduction in cardiovascular endpoints, we find no evidence that such treatment adversely affects patient-reported outcomes early in the course of the disease.
Using the endpoint assay, we found that the enzyme activity at pH 8 was 10 fold higher than at pH 5 and approximately 5 fold higher than at pH 9 (results not shown).
However, despite the availability of valid and reliable PCT friendly diagnostic and endpoint assessments, we found no suitable measure available for adverse event elicitation.
Analysing each endpoint separately, we found that statin use was associated with significantly lower risk of a recurrent DVT, HR 0.66 (95% CI 0.59 to 0.71), as well as recurrent PE, HR 0.87 (95% CI 0.78 to 0.97), compared to no statin use (table 4).
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com