Sentence examples for end of repeat from inspiring English sources

Exact(6)

First, the variation between households and businesses among the targets with at least one victimization, suggests that on average, households do better at avoiding subsequent crimes than managers of businesses: a smaller proportion of households are at the upper end of repeat involvement than is the case with businesses.

Other mutants used were the amorphic zw3M11-1, the partial loss-of-function armF1a (mutation leads to Arginine 394 being mutated to a Histidine) that reduces Arm's affinity toward TCF, and the strong hypomorph armXM19 that contains a stop codon at the end of repeat twelve (stop codon introduced after amino acid 680), deleting the entire C-terminal region preventing Wg signaling [24].

A trans-esterification (TES) reaction (yellow arrow 1) catalyzed by Cas1 joins the protospacer to the 5′ end of repeat 1.

One end of repeat contig having no linkage to any other contigs, usually representing linear chromosome telomere or linear plasmid end (Panel 1 and 2), 4).

(2) Another TES reaction (yellow arrow 2) joins the other strand of the protospacer to the 5′ end of repeat 1 on the bottom (minus) strand, resulting in the formation of a gapped DNA duplex.

This PCR fragment from strain 91130, which spans the DNA portion harboring part of mgtA at the end of repeat 1, IS 1634-CE, and part of ptsG at the beginning of repeat 2, was sequenced (GenBank/EMBL Accession No. AM183254) and found to have some minor discrepancies, i.e., a deletion of 8 n.t. and eight single nucleotide polymorphisms (SNP) compared to the same genomic portion from type strain PG1.

Similar(54)

In this study, we used self-assembled peptide hydrogels with repeating peptide sequences and bioactive motifs at the end of repeating sequences, which are collagen mimetic peptides (CMPs).

A battery of tests was administered at the end of repeated dosing (30 day examination) and 30 days after dosing ceased (60 day test).

While small sensitization effects are often detected soon after drug exposure, their magnitude increases over time [ 14- 16], indicating that neuroadaptations underlying sensitization continue to develop long after the end of repeated drug pretreatment.

Five patterns emerged from the interpretation of the raw RT-CGM data in the first phase of our analysis; four of these indicated a response to the technology, apparent in eAG0 versus mean glucose as of cycle 1 and lower A1C at the end of repeated RT-CGM cycles.

As this region was too large to PCR across, primers were designed from unique sequence into the repeat on both ends to confirm its position in the genome and from the end of the repeat to the beginning of the repeat to confirm that the repeat was present in tandem.

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