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non-homologous end joining breaks.
Microhomology-mediated end joining is also a mutagenic repair pathway.
Our data suggest that PLP1 deletions are likely caused by nonhomologous end joining.
DNA sequence analysis revealed that this deletion may be the result of microhomology-mediated end joining.
Three mechanisms exist to repair double-strand breaks (DSBs): non-homologous end joining (NHEJ), microhomology-mediated end joining (MMEJ), and homologous recombination.
The released free ends may undergo end joining processes.
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The contigs were assembled into scaffolds using paired-end joining.
DSBs can be repaired either by the homologous recombination or nonhomologous DNA-end joining (NHEJ) mechanism.
DNA-end joining is carried out by the NHEJ ligase complex LigIV/XRCC4/XLF.
DSBs can be repaired by two mechanisms, recombinatorial repair or nonhomologous DNA-end joining (NHEJ).
After paired-end joining and gap-filling, the contigs were subsequently assembled into scaffolds.
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