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The role of enteroviruses in encephalitis is not fully established.
But whether their levels in cerebrospinal fluid could be used as an auxiliary indicator in differentially diagnosing tuberculous meningitis and viral encephalitis is not yet clear.
Despite a better knowledge of pathophysiology, and a wider use of new molecular technologies for the diagnosis, the etiological diagnosis of acute encephalitis is not assessed in most cases.
Antibody-related limbic encephalitis is not always easy to diagnose but often responds well to treatment.
These findings suggest that anti-NMDAR encephalitis is not rare and likely commonly misdiagnosed as a seizure disorder or psychiatric illness [ 18].
Moreover, damage in patients with herpes simplex encephalitis is not limited to the perirhinal cortex but includes many other medial and lateral anterior temporal lobe regions.
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In particular, mesiotemporal hypermetabolism, which is typically seen in limbic encephalitis, was not observed.
The diagnostic assessment of patients with suspected encephalitis was not standardised.
The anti-tickborne encephalitis serum immunofluorescence titer rose from 1 80 (June 29) to 1 640 July 144); titers against dengue, West Nile, yellow fever, and Japan B encephalitis were not elevated.
The mechanisms by which TBEV causes encephalitis are not completely understood, but a composite of direct cytolytic viral damage and of considerable immune pathology is likely.
In humans, the symptoms are mainly non-specific and influenza-like, however, severe pneumonia, endocarditis, and encephalitis are not uncommon [ 15, 16].
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