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In an empty control room, boxy PCs sat on workstations against a wall.
The complementing plasmid and the empty control plasmid were passaged through RN4220 and then transformed into S. aureus strain USA300 ΔflipR.
After 3 weeks, the defects treated with α-CSH/ACP (60/40) showed more new bone formation (21.1%) than the empty control (13.6%) (p < 0.05).
In a rabbit defect model, the groups treated with PLCL scaffolds exhibited significantly enhanced cartilage regeneration compared to groups harboring an empty control or PLGA scaffolds.
After 52 weeks, all but one empty control defect were covered by bone and a very thin layer of cartilage (ICRS 7 points).
Accordingly, 293 cells were transfected with CEACAM1 or the empty control vector (pcDNA).
This plasmid, pEML1, and empty control were transformed into the S. pombe wild type and leu3Δ deletion strains.
The total amount of DNA added in each transfection was kept constant by addition of an empty control vector.
Note there was no interference of Hha-mediated inhibition of yeast agglutination with the empty control plasmid pACYC184 (Figure 3).
MB114 and SVEC cell lines were transduced with LCN7 encoding pMSCV viral particles or empty, control viral particles.
Single infection with an empty control virus or Nanog alone did not produce any colonies (Fig. 1B).
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