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Exact(14)
A secondary analysis (17 studies) that was based on the calculation of percent change values also revealed small correlations for changes in motor performance with agonist (r = − 0.25, 11 studies) and antagonist (r = − 0.24, 7 studies) EMG amplitude as well as agonist (r = 0.46, 8 studies) and antagonist (r = 0.29, 5 studies) EMG duration.
a Data points for the agonist EMG duration (8 studies, 14 data points).
In terms of EMG duration, duration of muscle activity was used.
b Data points for the antagonist EMG duration (5 studies, 5 data points).
Pre and post practice data for motor performance and EMG duration.
In other words, as motor performance improved agonist and antagonist EMG duration decreased, yet agonist and antagonist EMG amplitude increased.
Similar(46)
Further, practice-related alterations in motor performance were assessed along with associated changes in quantitative (i.e., EMG amplitude, duration) but not in qualitative (i.e., timing of muscle activity, amount of coactivation) myoelectric variables or other biomechanical variables (i.e., kinetics and kinematics).
The shorter EMG burst duration with reflex cough is modulated to maintain the elevated pressure without using too much pressure or losing the vital air needed to clear the airway over time.
In this case, a decrease in EMG amplitude and duration would be an adequate adaptation.
The present systematic review revealed small-sized correlations between practice-related changes in motor performance and agonist and antagonist EMG amplitude and duration in healthy individuals.
We found small associations (range r = 0.015 0.50) of practice-related changes in motor performance with measures of agonist and antagonist EMG amplitude and duration.
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