Sentence examples for embryonic regulation from inspiring English sources

Here we take advantage of these observations to design a molecular screen for new genes involved in the earliest stages of specifying embryo polarity; together with bioinformatic analysis and embryological experiments we identify the transcription factor Pitx2 as a direct and essential regulator of cVg1 expression both during normal development and in embryonic regulation (induced twinning).

We have performed the first detailed analysis of embryonic regulation of hepcidin and the role of hjv during embryonic development.

Our group has characterized embryonic regulation of the mouse hematopoietic niche, a key extrinsic component of the hematopoietic environment (Sugiyama et al., 2011a).

DOI: http://dx.doi.org/10.7554/eLife.03743.012 To determine whether Pitx2 is important for embryonic regulation and for controlling cVg1 expression, we used targeted electroporation of morpholino oligonucleotides (MOs).

DOI: http://dx.doi.org/10.7554/eLife.03743.008 > -wrap-foot>> -wrap-foot>> -wrap-foot> To determine the temporal relationship between Pitx2 and cVg1 in whole embryos and during embryonic regulation, we compared their expression in time-course.

Functional enrichment clustering using the DAVID resource resulted in a few significant groups, the most relevant of which included hemopoeisis and erythrocyte homeostasis, embryonic morphogenesis, regulation of cell cycle and cell differentiation, regulation of apoptosis, intracellular signaling, and a variety of signaling pathways (e.g., TPO, TGF-Beta, JAK-Stat, Wnt, ERBB, IL6 signaling).

MTA1 was found to play roles in circadian rhythm maintenance [ 106], embryonic development regulation, and visual performance by regulating rhodopsin expression [ 102].

Germ cell and embryonic gene regulation involve gene regulatory networks that integrate intercellular signaling and epigenetic regulation to reprogram chromatin state and produce activators and repressors required for pluripotency and induction of lineage commitment.

Previous studies on the loss of GRP94 function showed that it is required for embryonic development, regulation of toll-like receptors and innate immunity of macrophages.

bOther categories include: embryonic development, regulation of enzyme activity, growth, mesoderm development, pattern specification, coagulation, extracellular structure organization and biogenesis, pathogenesis, rhythmic process.

This opposes the hypothesis that a smaller meristem in Ler-0 might be the cause of increased sensitivity to zll mutations, but suggests that fundamental differences in embryonic meristem regulation may contribute to variable zll expressivity in these accessions.