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The strong correlation between H2BK123ub1 levels in coding regions and transcriptional activity suggests that the majority of H2BK123ub1 in the yeast genome is established during transcription elongation through the association of the H2B ubiquitylation machinery with elongating RNA polymerase II [ 32].
Bone elongation through callotasis is a relatively simple procedure from a surgical point of view.
The root system architecture was also recorded twice a week by tracing the root elongation through the transparent face of the root observation boxes.
Meanwhile, polyanionic cyclodextrin inhibited the shortening action with n-octanol, which has a strong ability to inhibit polymer elongation through an undetermined function.
However, free fatty acids can be produced by breaking the fatty acid elongation through the overexpression of an acyl-ACP thioesterase.
Farrell et al. (2014) and Farrell and Healy (2017) also show that pore elongation through cataclasis and shearing can create a permeability anisotropy adjacent to faults in porous sandstone.
We believe that slow and progressive elongation through external fixation helps avoid the sudden stretching of the sympathetic nerves located around the blood vessels, which are believed to be the main etiology of this complication [36, 37].
The basic steps in this process include chromatin decondensation, pre-initiation complex assembly, and RNA polymerase II (RNA pol II) elongation through the gene [1], [2].
It remains possible that the phosphorylated CSpt5 may have a role in positively regulating transcription elongation through recruiting active elongation complexes.
The ratio of expression from the downstream reporter compared to the control reporter upstream provides a measure of the relative rates of successful elongation through the intervening sequence.
If this is the case, it will be important to understand why sAPPβ stimulates axon elongation through Egr1 while it does not act in LTP [7].
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