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SINE insertions were enriched in regions related to neural phenotypes in mouse (abnormal spinal cord dorsal column morphology, P = 1.6 × 10−5; abnormal neural fold elevation formation, P = 1.3 × 10−) and to genes showing hindbrain expression (TS18_hindbrain, P = 1.1 × 10−4; TS21_hindbrain, P = 1.1 × 10−4).
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Two varieties of Carthamus tinctorius flowers (Jawhara and 104) were harvested randomly from two different Tunisian localities: Beja (North Western Tunisia; latitude 36° 431.19′19′′ (N); 9° 11′ 14.52′′ E; altitude 225 m) and Tunis (latitude 36° 50′ 29.68′′ (N); longitude 10° 12′ 19.44′′ (E); 3 m elevation) at bud formation (Bu), flower formation (F), full flowering (FF), and seed formation (Se).
Furthermore the screening assay is sufficiently sensitive to detect an elevation in macropinosome formation upon overexpression of SNX5, a known regulator of macropinosome formation [52].
Furthermore, we observed 2 distinct patterns (an increase and decrease) for the momentum of scapular upward rotation with arm elevation after knot formation.
Cutting forces, temperature elevation and chip formation were measured in real time for two different rake angles and six different cutting depths.
The aim of this study was to answer the following questions: (1) Does an increase in elevation and the formation of tree-fall gaps affect whether dispersed seeds in cloud forests are taken by predators, infected by pathogens or germinate?
Amylin precursor upregulation by MCP-1 may contribute to amylin elevation and amyloid formation.
Moreover a 3 fold elevation in macropinosome formation (p<0.05) was observed for cells overexpressing pEYFP-Rabankyrin-5 (Figure 2C).
Based on these findings, we investigated the relationship between intracellular calcium elevation and the formation of a tissue-tissue boundary between two distinct explants.
The observation that the protein-scaffolding SH3 domain of SNX18 is required for an elevation in macropinosome formation may suggest that an intact association between SNX18 and other key molecular effectors is required in driving macropinocytic uptake.
Our findings that coexpressing PTEN(G129E) and SNX9 or SNX18 was synergistic in the elevation of macropinosome formation support this model, although further work is needed in characterizing potential interactions between PI(3)K and SNX9 and/or SNX18.
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