Sentence examples for elevated urea from inspiring English sources

Both elevated urea and creatinine of the serum treated with STZ can be observed in both the figures and normalized range of these kidney enzymes were acquired by treating the model with 1.5% EO.

Furthermore, elevated urea concentrations had an effect of decreasing uterine pH during the luteal phase [ 18- 20].

Elevated urea concentrations stood out above glucose, sodium and potassium concentrations as a strong independent predictor of mortality.

Nine of 19 patients tested (47%) had an elevated leukocyte count (>10 ×10/L), and 11 (61%) of 18 had elevated urea levels (>7 mmol/L).

Laboratory abnormalities included mild nonregenerative anemia, mature neutrophilia (23,000/μL, reference range 3,600 11,500/μL), mild hypoalbuminemia, and mildly elevated urea and creatinine levels.

Increased risk for developing delirium was found for cognitive and physical impairment, elevated urea nitrogen level, and the number of leucocytes.

All the 15 of the 116 (12.9%) patients with altered sensorium manifested one or more metabolic abnormalities [hyponatremia (n = 9); hypokalemia (n = 7); hyperbilirubinemia (n = 3); elevated transaminases (n = 12) elevated blood urea (n = 11); and elevated serum creatinine (n = 3)] or type II respiratory failure and carbon dioxide retention (n = 11).

Elevated blood urea nitrogen (BUN) at high temperatures was a good indicator of gill osmoregulatory failure.

Animals without TEC had statistically significant hyponatremia, elevated serum urea nitrogen, and lower stool short chain fatty acids (13.5 μmol/kg vs 84.2) with an abnormal distribution.

In the immediate period after beginning treatment, there was an impairment of renal function (elevated serum urea, creatinine and osmolality; increased fractional excretions of water, sodium and osmotic particles), with a return to normal by 10 days postnatally.

Blood assays show Hawaiian turtles with FP have elevated blood urea nitrogen compared to disease free turtles [64] which in the absence of gastrointestinal pathology [60] can indicate enhanced dietary intake of N [65].