Sentence examples for electrical dysfunctions from inspiring English sources

The induction of DCM was evidenced by cardiac pump and electrical dysfunctions.

we demonstrated that the ER stress sensors, referred as PERK, IRE1 and ATF6, were activated in ROS- mediated ER stress- induced cell apoptosis in rat model of DCM which was characterized by cardiac pump and electrical dysfunctions.

This study suggests a new recondition of signaling transduction in explaining mechanism of DCM: 1. ROS- induced ER stress mediated myocyte apoptosis may play an important role in DCM which is characterized by cardiac pump and electrical dysfunctions.

This consensus report revised the definition of cardiomyopathy to include "mechanical or electrical dysfunction that usually exhibits inappropriate ventricular hypertrophy and dilatation due to a variety of causes that frequently are genetic".

Here, we discuss some of the key cellular and network electrophysiological changes that are associated with ischemic injury and highlight the link between alterations in the energetic/metabolic substrate of the heart and electrical dysfunction.

Cardiomyopathy is a diverse group of cardiac disorders characterized by mechanical and/or electrical dysfunction of the cardiac muscle.

Electrical dysfunction of the voltage-sensitive ion channel is associated with potentially lethal ventricular arrhythmias in humans.

Thus, the cellular increase in Ca2+, which is the reason for its extracellular decrease, promotes NADH hyperoxidation and electrical dysfunction after anoxia in hippocampal slices [ 56].

Given the arrhythmogenic implications of F-M coupling discussed above, a thorough understanding of F-M coupling could provide insights into the mechanisms of electrical dysfunction during cardiac fibrosis.

Fibroblasts play a significant role in the development of electrical dysfunction of the heart during disease states such as myocardial infarction and various cardiomyopathies; however the mechanisms are only partially understood.

Additionally, increased late INa,L may result in Ca2+ induced electrical cardiomyocyte dysfunction as shown in SCN5A-∆KPQ knockin mice resembling the Long-QT3 syndrome [ 77].