In order to characterize the effect of CTF correction on the preservation of structural integrity, the data was either corrected for CTF from estimated defocus, or by applying arbitrary defocus values (Figure 3).
The hardness values are either corrected for the indentation size effect or following Oliver Pharr model.
First, since studies of multiple SNPs and multiple phenotypes create the potential for multiple comparison problems and frequent false positive results, it would be important to either correct for multiple comparisons or know the predicted false discovery rate.
Each phenotype was either corrected for environmental factors (e.g. year of birth or farm) and rescaled by correcting for the overall mean (traits 3, 4, and 5) or was a rescaled, weighted mean of corrected progeny phenotypes (traits 1 and 2), for which many animals have no individual performance data.
M30 antigen levels, either corrected for CK18 or uncorrected, did not correlate with overall survival, disease-free survival or disease recurrence, in contrast to caspase-3 activity, indicating the importance of non-epithelial caspase-3 activity, that is, stromal apoptosis.
Controls did not activate this perigenual rACC region for either correct or error responses.
They should either be corrected for the stage of pregnancy, or total thyroxine levels should be used instead for diagnosis.
The training effect for repeated testing can either be corrected for, or an extra training trial can be given at the beginning.
Finally, Schmiedek et al. [34] reported that correcting for either individual or group means on a reaction time-based estimate of IIV may lead to incorrect inferences.
In order to disentangle a possible selective effect from BGC, we analyzed SNP allele frequencies in noncoding genomic sequences after correcting for either GC content or recombination rate.
