Sentence examples for efficient tissue repair from inspiring English sources

It is perhaps these differences which contribute to efficient tissue repair seen in the clinic with fetal cell therapy.

Insights into the molecular origins of cartilage lubrication could lead to more efficient tissue repair and to longer-lasting prostheses for hip and knee joints.

For in vivo studies, we employ rodent models of volumetric skeletal muscle loss, cardiotoxin and BaCl2 injury as well as myocardial infarction and transverse aortic constriction to study how cell, tissue engineering, and gene (viral) therapies can lead to safe and efficient tissue repair and regeneration.

In addition, some senescent cells secrete molecules such as growth factors, pro-inflammatory cytokines, and proteases, among others that, although essential for efficient tissue repair, also damage the organism.

When migrating cells arrive at a point where the cytokine concentration reaches a precise threshold they switch from a migrating phenotype to a proliferating one, leading, in the case of wound healing example, to an efficient tissue repairing.

Many studies revealed that platelet concentrates for surgical use can be used as efficient adjuvants for tissue repair [ 1– 5].

Overall, the CBrhTE scaffold could synchronously improve all three of the basic components required for cartilage tissue engineering in vitro, which paves the road for designing and building more efficient tissue scaffolds for cartilage repair.

A deeper understanding of the functional properties of the cellular microenvironment thus emerges as an important prerequisite for the development of more efficient therapeutic approaches in tissue repair and regeneration and diseases ranging from chronic and acute inflammation to cancer.

Although the ideal parameters and type of electrical stimulation that is safe and efficient in promoting connective tissue repair, particularly cartilage repair [ 33, 44], have not been established, studies have demonstrated that low frequencies seem to be more efficient in promoting the repair of different connective tissues since they act by altering the membrane potential of the cell [ 25, 32].

The lack of graft-host electrical integration is therefore a critical obstacle to development of efficient tissue engineering therapies for heart repair.

In addition, further questions in the field will be pondered, including the phenotypic and functional overlap between pericytes and culture-derived MSC, and the concept of pericytes as efficient producers of differentiation factors to speed tissue repair.