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Several studies reported neurological side effects of these molecules [33, 34].
These concerns have led to intense research studies on the effects of these molecules on various biological systems.
While clinical trials for general ABC transporters such as verapamil have been performed, these studies were ended due to the dose-limiting toxic side effects of these molecules [32].
The addition of surfactants (utilizing ionic and nonionic surfactants as model compounds) results in a significant reduction of coarsening rates, in addition to known effects of these molecules on elemental sulfur solubility.
In order to assess the potential of an HA/SP4.2 modified BAM to influence cellular functions associated with bladder healing, experiments were conducted to evaluate the individual and combined effects of these molecules on in vitro fibroproliferative endpoints within a co-culture model.
We therefore decided to compare the differential effects of these molecules in the induction of dendritic filopodia.
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Next, the effect of these molecules on the stability of a quartz suspension was investigated.
We are in the process of studying the biological effect of these molecules on retinoic acid signaling pathways.
However, a synergistic or antagonistic effect of these molecules may play an important role on the observed inhibition efficiency of CA extract.
The ultimate effect of these molecules is determined by the intrinsic antioxidant capacity of cancer cells as the cytotoxic potential of these agents is lost upon antioxidant co-treatment [252 254].
We report on the possible effect of these molecules on Pt, Pd and Pt Pd model catalysts at lower temperatures and somewhat higher iodine or iodide concentrations, as compared to inferred catalyst operational parameters in a reactor building during a severe accident.
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