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The inhibitory effects of PC on the cell proliferation were detected by MTT assay.
The decreased proteins such as HSP60, nucleolin, PPase, peroxiredoxin-4 and the increased protein (mtSSB) were identified in SKOV-3 cells after PC treatment, indicating that the effects of PC on tumor cell apoptosis may be relate to multiple target proteins.
Several studies, predominantly from the USA, have shown positive effects of PC on health outcomes [ 5, 14].
Since PC treated cells showed a significant reduction in colony formation ability, we next sought to determine the effects of PC on the migration behavior of breast cancer cells.
A study of the effects of PC on the self-care behaviours of newly diagnosed DM patients revealed that the treatment was highly appreciated and even after 9 months improvements in eating and exercising habits were seen.
Since tissue invasion and metastasis are the main causes for mortality in triple negative breast cancer [ 30], we investigated the effects of PC on cellular migration and aggregation behavior of MDA MB 231.
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While the positive effects of PS on cognitive appraisals did not extend to the affective stress response directly or indirectly, the negative effect of PC on the affective stress response was fully mediated by the secondary self-confidence appraisal.
The effect of PC on WI is found very little here again.
Next, we examined the effect of PC on cell proliferation using primary OA FLSs.
The downregulatory effect of PC on these genes indicates that PC may reverse the processes of terminal differentiation and biomineralization.
There is no significant effect of PC on normal Human Breast MCF 10A cells indicating its non-toxicity to normal cells.
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