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In this study, two doses of LP were given to ovariectomized rats for three different time intervals; 3, 6 and 9 weeks to evaluate the dose and time-dependent effects of LP on bone biomechanical strength.
We then look at the effects of LP on ST and NE, and then the effect of ST on NE is examined.
Phytoestrogens are often potent antioxidants and anti-inflammatory agents which may explain the effects of LP on women's health.
Following these positive results of LP on bone microarchitecture, our present study was performed to evaluate further the effects of LP on bone strength.
The present study was therefore arranged to investigate the protective effects of LP on lungs against CCl4-induced oxidative damage in rats.
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We could not detect any effect of LP on IL-6 or IL-10 (Fig. 5).
However, the anti-MIMP deprived the effect of LP on the reduction of EPEC adhesion (P < 0.05, Figure 1D).
Therefore, these estimates can be interpreted as the causal effects of Lp(a) on PAD/ABI.
This study aimed to determine the effects of LP supplementation on bone biomechanical strength of postmenopausal osteoporosis rat model.
The pathophysiological effects of Lp(a) on atherosclerotic CVD can be broadly categorized into two aspects, named proatherogenesis and prothrombosis or antifibrinolysis.
As a consequence, OxPL/ApoB-enriched LDL is considered significantly responsible for the adverse effects of Lp(a) on cardiovascular system.
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