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Moreover, we investigated the regulatory effects of ID on the activation of MAPKs.
To evaluate the effects of ID on the inflammatory mediator production, blood samples were collected and analyzed using the ELISA method.
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Other studies have explored the effects of IDs on neutrophil activation, adhesion and/or migration.
To assess the effects of IDs on the adhesiveness of activated neutrophils independently from the activation of endothelial cells, we used hECM as a binding substrate.
Therefore, we evaluated the effect of ID on the phosphorylation of MAPKs.
Moreover, the effect of ID on compound 48/80-induced 48/80-inducedshock wanaphylacticted in a moushockdel.
Therefore, the effect of ID on the degree of cell viability was first examined using an MTT assay.
The inhibitory effect of ID on anaphylactic shock produced by compound 48/80 was evaluated using BALB/c mice.
In this study, we demonstrated the regulatory effect of ID on allergic reactions in vivo and in vitro.
Because NF-κB is a pivotal transcription factor of inflammatory and immune reactions [ 22], the effect of ID on the activation of NF-κB was investigated.
In this study, we evaluated the effect of ID on DNFB-induced AD-like skin lesion and anaphylactic shock in mouse models.
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