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We estimated the overall and conditional effects of FA on changes in weight and disease progression by one or more WHO stages.
For that reason, the present study investigated the long-term effects of FA on motor behavior and dopaminergic circuitry.
Specimens with four FA/GGBFS weight ratios (0, 1/9, 3/7 and 1) are prepared to estimate the effects of FA on the engineering performance.
Therefore, we first performed experiments to see the effects of FA on the fluorescence staining and the organization of two types of putative lipid raft markers (GM1, a lipid marker, and CD59, a protein marker).
Since imaging studies of cold-induced effects on lipid rafts require formaldehyde (FA) pre-fixation for immune staining of lipid raft-enriched Jurkat T cells, we first examined effects of FA on the fluorescence staining of various types of molecules in plasma membranes.
The effects of FA on hyperplastic mammary lesions and mammary carcinomas were determined in a neu-induced rat model.
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The ontology term and pathways analysis for FA exposed worms provided convincing support for an effect of FA on such production costs.
We further assessed the effects of FAs on ERα alone and ERβ alone in HeLa cells.
We have explored possible mechanism(s) for the effects of FAs on ER signaling by molecular modeling.
Figure 2.I. shows the effects of FAs on ERα (A) and ERβ (B) recruitment to the pS2 gene promoter.
These FAs also attenuated the effects of E2 under co-incubation conditions (Fig. 3.I.B). Figure 3.I shows the data for the effects of FAs on ERE-luciferase activity at a FAs concentration of 10−6 M and co-incubation with 10−9 M E2 (full data in Fig. S4).
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