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Taken together, results offer insights into the adverse effects of EA on daily well-being and suggest that EA is a context-specific regulatory strategy that might be best captured using a state-dependent measure.
In this regard, we have shown that the opioid system in the rVLM including endorphin and enkephalins, that largely act through stimulation of μ- and δ-opioid receptors, respectively, contributes importantly to the sympathoinhibitory effects of EA on pressor reflex19.
We examined the effects of EA on the survival rate of mice, inflammatory cytokine and pro-inflammatory mediator production, histopathological changes and protein expression of COX-2 in tissue sections from lung, liver and kidney.
Most studies of EA have used trait measures without considering the effects of EA on psychological functioning in naturalistic settings.
In addition, other signaling pathways may also contribute to the cytotoxic effects of EA on various cell types.
To assess the inhibitory effects of EA on Wnt/β-catenin signaling in CLL cells, real-time PCR was employed to detect the expression of some Wnt target genes.
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The present study was designed to investigate whether electroacupuncture (EA) was beneficial to extenuate the behavioral deficit in a rat model of depression induced by chronic unpredictable stress (CUS) and to observe the effect of EA on progenitor cell proliferation in the dentate gyrus (DG) of hippocampus.
The present study includes a series of cross-sectional studies designed to accomplish three goals: (1) to provide additional validation data for the BI-AAQ, (2) to assess the potential indirect effect of EA on the relationship between body image dissatisfaction and disordered eating cognition and behavior, and (3) to compare the BI-AAQ to an existing measure of body avoidance.
The stimulatory effect of EA on stress-induced delayed gastric emptying is mediated via cholinergic pathways.
The inhibitory effect of EA on stress-induced acceleration of colonic transit was not affected by guanethidine (GC=4.6+/-0.3 GC=4.6+/-0.3
The effect of EA on positive affect was not accounted for by the amount of negative affect experienced.
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