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The present study was designed to examine the effects of AF on endothelial function in human subjects.
In this study, we evaluated the effects of AF on IL-1β-stimulated HK-2 cells.
Importantly, the effects of AF on the memory cell subset were not associated with any detectable immune impairment in vivo.
We first aimed at confirming these effects of AF on sorted CD4+ T-cell subpopulations isolated from a cohort of uninfected human donors.
It was therefore thought worthwhile to investigate the effects of Af on neurodegenerative diseases, effects that have not been studied to date.
The complex relationship between AF, systemic inflammation, as well as endothelial/microvascular dysfunction and the effects of AF on ventricular calcium handling and oxidative stress are also addressed.
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The acceleration effect of AF on methane hydrate formation was concertedly concluded through experimental study on kinetics and theoretical analysis of heat transfer.
This was to avoid the potential effect of AF on MSNA.
There are few data on the effect of treatment of AF on mortality in ICU patients.
In a separate set of experiments, the effects of AF-16 on ICP were recorded.
The mechanisms behind the effects of AF-16 on brain tissue are, however, unclear.
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