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In the complex flow considered here – nonhomogeneous and anisotropic turbulence at moderate Reynolds numbers (<15,000) – with weak turbulence intensity (about 10%), it can be demonstrated that turbulent breakup mechanisms largely dominate mean flow effects; elongation and shear effects are shown to have minor effects on the breakup mechanisms.
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Within the elastomeric range, the PIB span molecular weight was the dominant factor effecting elongation at break.
It could be that we did not observe substantial effects on elongation because the energetics of curved to straight transitions on the microtubule end are not rate-contributing for elongation, perhaps because these transitions occur subsequent to end binding.
Effects of elongation on the electric resistance of graphite EVA ethylene-acetate copolymer) systems were studied.
A hypothetic mechanism is assumed to explain the effects of elongation and shear rates on the changes of conformation of polymer chain.
We do not believe that an inherent feature of Eμ-Myc lymphomas make them particularly sensitive to the effects of elongation inhibitor/Dxr combination since Eμ-Myc/Bcl-2 lymphomas showed significant resistance to these drug combinations (Fig. 2B).
This raises the possibility that the ability of Tat-SF1 to associate with the U2 snRNP, perhaps mediated by the snRNA's secondary structure, is responsible for the effects on elongation [1].
MLR confirmed that the presence of nonwoven textile had significant negative effects on elongation, deformation, and tensile work (P < 0.001 for all) and significant positive effects on tensile strength, puncture work, and puncture strength (P < 0.01 for all).
We focused on the effect of pulsed light laser irradiation vis-à-vis two distinct biological effects: neurite elongation under NGF stimulus on laminin-collagen substrate and cell viability during oxidative stress.
We focused on the effect of pulsed light laser irradiation in two distinct biological effects: neurite elongation under NGF stimulus on a laminin-collagen substrate and mitochondria membrane potential and activity under basal conditions and after oxidative stress.
Downstream of the 25 min transition point, the intrinsic acceleration of Pol II has completed, and additional positive effects on elongation rate by H3K79me2 may no longer be needed.
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