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To date, there is no known report of the effect of SM on paraspinal muscle excitability.
A positive effect of SM on germ cell differentiation was also observed in undifferentiated (Kit-negative) spermatogonia, in which RCCS conditions stimulate the expression of Kit and Stra8.
The effect of SM on meiotic entry of differentiating spermatogonia was confirmed by the increased expression at the mRNA and/or protein level of genes important for commitment and entry into meiosis (such as kit, scp3 and stra8) and of genes essential for early meiotic DNA recombination (such as spo11, scp3 and scp1).
The review suggested both a local and regional effect of SM on pain reduction.
However, the effect of SM on RIP1 is likely due to the degradation of cIAP1/2, as the effect of SM on TNF α production was recapitulated in cIAP1-knockout cells but not XIAP-deficient cells.
However, no previous study has examined the effect of SM on damage to the white matter and hippocampus induced by chronic cerebral hypoperfusion.
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In the present study we did not quantify changes in muscle activity following SM, but rather assessed the effects of SM on the evoked short-latency stretch reflex amplitude.
Inhibitory effects of SM on U87 cells proliferation in vitro and in vivo were more effective than that of temozolomide (TMZ), and SM has synergistic effects with TMZ in the glioma therapy.
The toxic effects of SM on the haematopoietic system are dose dependent, and it is concluded that SM causes aplastic or ineffective haematopoiesis.
Specifically, we quantified the effects of SM on the motor evoked potential (MEP) and short-latency stretch reflex amplitude of the erector spinae muscles.
We also directly assessed the effect of SM caller on the observed enrichment by comparing the SM calls made using two SM calling algorithms (Hydra [ 35] and Meerkat [ 36]) in two cancers studied in The Cancer Genome Atlas, namely breast invasive carcinoma ("BRCA") and Lung Squamous Cell Carcinoma ("LUSC").
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