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Three single crystal superalloys were designed based on the composition of superalloy CMSX-4 and effect of Re on the thermo-stability of these designed alloys has been investigated.
Our expression data also provides supporting evidence for the inhibitory effect of RE on muscle mitochondria functional activity.
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Effect of Res on the malignant transformation of CRC cells was examined by soft agar colony formation assay.
The inhibitory effect of Res on KITLG expression was abolished when silencing the endogenous miR-34c (P < 0.05, Fig. 2d).
Res increased miR-34c expression both in p53 + and p53 − CRC cells, suggesting the effect of Res on miR-34c expression was p53 independent.
Note that, Res arrested HCT-116 cells in G0/G1 phase while HT-29 cells in G2/M phase, indicating that the effect of Res on the cell cycle arrest was cell type dependent.
However, it was worth noting that p53 was elevated after treated with Res in HT-29 cells and, simultaneously, the inducement of miR-34c by Res was more prominent in HT-29 cells, indicating p53 facilitated the effect of Res on miR-34c expression.
The effects of RE on microstructure and micro-hardness of coatings were investigated.
The effects of RE on the microstructure and properties of laser coatings have also got more and more attention.
These findings are important in understanding the effects of Re on the microstructure and providing guidance on the design of Mo Re Si alloys.
The present investigation was designed to examine the possible effects of RE on hypothalamic concentrations of DA, NE, 5HT and their major metabolites homovanillic acid (HVA), 3-4-dihydroxyphenyl acetic acid (DOPAC), 3-methoxy mandelic acid (VMA) and 5-hydroxyindole 3-acetic acid (5HIAA), respectively.
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