Sentence examples for effect of inflammation and from inspiring English sources
To discriminate between an effect of prematurity from an effect of inflammation, and to document any improvement in survival, mice were killed at 3, 6, and 16 hours after injection.
It was concluded that pulsed electromagnetic fields are able to stimulate the articular cartilage in relation to the catabolic effect of inflammation and subchondral bone marrow edema, and to promote osteogenic activity at the necrotic area to prevent trabecular fracture and subchondral bone collapse.
Moreover, our findings suggest the possibility of a mutually potentiating effect of inflammation and hyperglycemia in cardiovascular risk prediction.
The seizure-promoting effect of inflammation and its influence on the outcomes of SE patients has been demonstrated in recent studies.
By adjusting by CRP, a proinflammatory biomarker, we controlled for the confounder effect of inflammation, and thus, the association between ferritin levels, as a marker of excessive body iron stores, and the metabolic syndrome was strengthened.
Vaccine- and placebo-associated changes in diastolic and systolic blood pressure were used as co-regressors in both the analysis of the main effect of inflammation and between subject correlations.
Hence, an experimental inflammation manipulation was applied that avoided this confound, isolating effects of inflammation and social interaction.
The increased prevalence of glaucoma in chronic uveitis reflects the accumulated detrimental effects of inflammation and probably the consequence of chronic corticosteroid therapy on an initially normal trabecular meshwork [15, 16].
Assessing the flow density of the deep vascular complex may provide more complete information regarding the deleterious effects of inflammation and may provide an additional biomarker of inflammatory activity that may be quantified to assess progression and response to therapy.
The role of soluble factors, drug transporters, microvesicles, as well as the importance of direct cell cell contact, in addition to the effects of inflammation and immune surveillance in microenvironment-mediated drug resistance are discussed.
The substantia nigra (SN), a CNS region affected in PD, is particularly susceptible to inflammatory insults and possesses the highest density of microglial cells, but the effects of inflammation and in particular TNF-α on neuronal survival in this region remains controversial.
