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The mechanisms underlying the aggravating effect of hyperglycemia on brain damage are still elusive.
This study was designed to investigate the effect of hyperglycemia on tissue-fixed macrophages (MØ) PGE2 production.
In contrast, our results and other results also did not obtain macrosomic fetuses when studying the effect of hyperglycemia on offspring.
Furthermore, we explicitly studied the effect of hyperglycemia in comparison to normoglycemia, while glycemia was maintained between 63 and 108 mgmol · dL-1 in that study.
This study was designed to investigate the effect of hyperglycemia and angiotensin II (AngII) on renal hypertrophy and proteinuria in the pregnant diabetic rat.
However, the effect of hyperglycemia in combination with high nitric oxide (NO) level (reported to be increased during glucotoxicity), on brain functions is not clear yet.
To identify the frequency of hyperglycemia in children who are nondiabetic and critically ill and assess the independent effect of hyperglycemia on outcome.
The apoptotic effect of hyperglycemia and DEDTC on INS-1E cells was tested next.
Even though olfactory dysfunction is indicated during diabetes, little is known about the effect of hyperglycemia on the olfactory system.
The mechanism by means chronic diabetes mitigates the effect of hyperglycemia in acute cardiovascular event is unknown.
While olfactory dysfunction is indicated in diabetes, the effect of hyperglycemia on olfactory receptor neurons (ORNs) remains unknown.
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