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There have also been studies showing the apoptotic effect of COS on human hepatocellular carcinoma cells by the upregulation of Bax (Bcl-2-associated X protein) [ 87].
Even though the amylase inhibitory effects seen in COS leaf is marginal in the current study in agreement with the previous study [ 45], the current study reveals a significant inhibitory effect of COS on glucosidase activity.
The effect of COS on GT densities in A. annua is undocumented in the literature, but Lei et al. (2011) reported a significant increase in both AN and HP levels in leaves after foliar application of COS.
Senevirathne et al. [ 91] studies on the effect of COS on tert-butyl hydroperoxide (t-BHP) induced damage in Chang liver cells showed that COS protected Chang liver cells against oxidative damage induced by t-BHP via inhibiting production of ROS and lipid peroxidation and the elevation of the levels of antioxidant enzymes.
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The effect of CO on EP was equivocal.
The activity of Pt/Ba/Al was greatly depressed when using CO as reductant at low temperatures owing to the strong poisoning effect of CO on Pt.
Glasses composed of (P2O5 45(Na2O)20(TiO2)05(CaO)30 − x(CoO)x(x = 0, 5, 10, and 15 mol%) were fabricated and the effect of Co on physicochemical properties including density, glass transition temperature (Tg), degradation rate, ion release, and pH changes was assessed.
The results of this large study are consistent with an independent effect of CO on mortality.
However, the effect of CO on RANKL-induced osteoclastogenesis has not been investigated.
Sodium nitroprusside (SNP) was used to assess the effect of CO on vasorelaxation by a nitrite-independent NO donor.
The effect of CO on total and cardiovascular mortality was observed in western and southern European cities, whereas there was essentially no CO effect in eastern cities.
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